Studies on the antiarrhythmic actions of prostaglandins
Graduate
Membrane actions of antiarrhythmic drugs
Graduate
Therapeutic interventions in acute myocardial infarction. Survey of the ACCP Section on Clinical Cardiology
Studies on the metabolism of tocainide in humans
Graduate
Therapeutic trends in the management of patients with acute myocardial infarction (1975-1984): the Worcester Heart Attac...
As part of an ongoing community-wide study of time trends in the incidence and case-fatality rates of patients hospitalized with acute myocardial infarction (MI) in all 16 Worcester, Massachusetts, metropolitan hospitals during the years 1975, 1978, 1981, and 1984, changes over time in the therapeutic management of 3263 patients with validated acut...
A community-wide perspective of secular trends in the therapeutic management of patients with acute myocardial infarctio...
As part of an ongoing community-wide study of time trends in the incidence and case-fatality rates of patients hospitalized with acute myocardial infarction (MI) in 16 Worcester, Mass., metropolitan hospitals during the calendar years 1975, 1978, 1981, and 1984, changes over time in the therapeutic management of 3,263 patients with validated acute ...
Pharmacological and antiarrhythmic properties of quinacainol : a new sodium channel blocker?
Graduate
Antifibrillatory actions of K+ channel blocking drugs
Graduate
Adherence to national guidelines for drug treatment of suspected acute myocardial infarction: evidence for undertreatmen...
...
BACKGROUND: Evidence-based guidelines for the treatment of patients with acute myocardial infarction (AMI) have been published and disseminated by the American College of Cardiology and the American Heart Association. Few studies have examined the rates of adherence to these guidelines in eligible populations and the influence of age and gender on ...
Ethylisopropylamiloride Diminishes Changes in Intracellular Na, Ca and pH in Ischemic Newborn Myocardium
Numerous studies suggest that in adult hearts myocardial ischemic injury is in part the result of proton stimulation of Na/H exchange which increases intracellular Na (Nai) and thus leads to increases in intracellular Ca concentration ( [Ca]i) due to changes in Na/Ca exchange flux. Corollary to the hypothesis, inhibition of Na/H exchange diminishes...
