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Correction: The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical per...

Gomes, Felipe V Zhu, Xiyu Grace, Anthony A

A correction to this paper has been published and can be accessed via a link at the top of the paper.

Heterogeneity matters: implications for Poeppl et al.'s (2019) meta-analysis and future neuroimaging research on psychop...

Latzman, Robert D Patrick, Christopher J Lilienfeld, Scott O

Cortical surface area alterations shaped by genetic load for neuroticism.

Opel, Nils Amare, Azmeraw T Redlich, Ronny Repple, Jonathan Kaehler, Claas Grotegerd, Dominik Dohm, Katharina Zaremba, Dario Leehr, Elisabeth J Böhnlein, Joscha ...

Published in Molecular psychiatry

Neuroticism has been shown to act as an important risk factor for major depressive disorder (MDD). Genetic and neuroimaging research has independently revealed biological correlates of neurotic personality including cortical alterations in brain regions of high relevance for affective disorders. Here we investigated the influence of a polygenic sco...

Correction: Common-variant associations with fragile X syndrome.

Crowley, James J Szatkiewicz, Jin Kähler, Anna K Giusti-Rodriguez, Paola Ancalade, NaEshia Booker, Jessica K Carr, Jennifer L Giamberardino, Stephanie N Crawford, Greg E Losh, Molly ...

Published in Molecular psychiatry

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

Glucose disturbances, cognitive deficits and white matter abnormalities in first-episode drug-naive schizophrenia

Zhang, Xiangyang Yang, Mi Du, Xiangdong Liao, Wei Chen, Dachun Fan, Fengmei Xiu, Meihong Jia, Qiufang Ning, Yuping Huang, Xingbing ...

Published in Molecular Psychiatry

Disturbance of glucose metabolism may be implicated in cognitive deficits of schizophrenia in its early phases. Many studies have reported the important role of widespread disruption of white matter (WM) connectivity in pathogenesis, cognitive deficit and psychopathology of schizophrenia. However, no study has investigated their inter-relationships...

The pathophysiological impact of stress on the dopamine system is dependent on the state of the critical period of vulne...

Gomes, Felipe V. Zhu, Xiyu Grace, Anthony A.

Published in Molecular Psychiatry

Unregulated stress during critical periods of development is proposed to drive deficits consistent with schizophrenia in adults. If accurate, reopening the critical period could make the adult susceptible to pathology. We evaluated the impact of early adolescent and adult stress exposure (combination of daily footshock for 10 days and 3 restraint s...

Reply to Veronese and Smith: Healthy dietary indices and risk of depressive outcomes: a systematic review and meta-analy...

Lassale, Camille Batty, G David Akbaraly, Tasnime

Published in Molecular psychiatry

Correction: Cellular and molecular basis for stress-induced depression.

Seo, J-S Wei, J Qin, L Kim, Y Yan, Z Greengard, P

Published in Molecular psychiatry

An amendment to this paper has been published and can be accessed via a link at the top of the paper.

Predictors for a dementia gene mutation based on gene-panel next-generation sequencing of a large dementia referral seri...

Koriath, C Kenny, J Adamson, G Druyeh, R Taylor, W Beck, J Quinn, L Mok, T H Dimitriadis, A Norsworthy, P ...

Published in Molecular psychiatry

Next-generation genetic sequencing (NGS) technologies facilitate the screening of multiple genes linked to neurodegenerative dementia, but there are few reports about their use in clinical practice. Which patients would most profit from testing, and information on the likelihood of discovery of a causal variant in a clinical syndrome, are conspicuo...

Early restoration of parvalbumin interneuron activity prevents memory loss and network hyperexcitability in a mouse mode...

Hijazi, Sara Heistek, Tim S Scheltens, Philip Neumann, Ulf Shimshek, Derya R Mansvelder, Huibert D Smit, August B van Kesteren, Ronald E

Published in Molecular psychiatry

Neuronal network dysfunction is increasingly recognized as an early symptom in Alzheimer's disease (AD) and may provide new entry points for diagnosis and intervention. Here, we show that amyloid-beta-induced hyperexcitability of hippocampal inhibitory parvalbumin (PV) interneurons importantly contributes to neuronal network dysfunction and memory ...

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