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Upregulation of pentraxin-3 in human endothelial cells after lysophosphatidic acid exposure.

Authors
  • Gustin, Cindy
  • Delaive, Edouard
  • Dieu, Marc
  • Calay, Damien
  • Raes, Martine
Type
Published Article
Journal
Arteriosclerosis Thrombosis and Vascular Biology
Publisher
Ovid Technologies Wolters Kluwer -American Heart Association
Publication Date
Mar 01, 2008
Volume
28
Issue
3
Pages
491–497
Identifiers
PMID: 18162608
Source
Medline
License
Unknown

Abstract

Our findings favor the proatherogenic role of LPA, a bioactive lipid produced by activated platelets and present in oxLDL, because it enhances pentraxin-3 secretion that could contribute to the accumulation of monocytes in the atherosclerotic lesion.

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