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Targeting the SMO oncogene by miR-326 inhibits glioma biological behaviors and stemness.

Authors
  • Du, Wenzhong1
  • Liu, Xing1
  • Chen, Lingchao1
  • Dou, Zhijin1
  • Lei, Xuhui1
  • Chang, Liang1
  • Cai, Jinquan1
  • Cui, Yuqiong1
  • Yang, Dongbo1
  • Sun, Ying1
  • Li, Yongli1
  • Jiang, Chuanlu1
  • 1 Department of Neurosurgery, Second Affiliated Hospital of Harbin Medical University, Harbin, China (W.D., X.L., Z.D., X.L., L.C., J.C., Y.C., D.Y., Y.S., Y.L., C.J.); Department of Neurosurgery, Huashan Hospital, Fudan University, Shanghai, China (L.C.). , (China)
Type
Published Article
Journal
Neuro-Oncology
Publisher
Oxford University Press
Publication Date
Feb 01, 2015
Volume
17
Issue
2
Pages
243–253
Identifiers
DOI: 10.1093/neuonc/nou217
PMID: 25173582
Source
Medline
Keywords
License
Unknown

Abstract

This work suggests a possible molecular mechanism of the miR- 326/SMO axis, which can be a potential alternative therapeutic pathway for gliomas.

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