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Spectrum and frequency of cardiac channel defects in swimming-triggered arrhythmia syndromes.

Authors
Type
Published Article
Journal
Circulation
1524-4539
Publisher
Ovid Technologies Wolters Kluwer -American Heart Association
Publication Date
Volume
110
Issue
15
Pages
2119–2124
Identifiers
PMID: 15466642
Source
Medline
License
Unknown

Abstract

In contrast to previous studies that suggested universal LQT1 specificity, genetic heterogeneity underlies channelopathies that are suspected chiefly because of a near-drowning or drowning. CPVT1 and strategic genotyping of RyR2 should be considered when LQT1 is excluded in the pathogenesis of a swimming-triggered arrhythmia syndrome.

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