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Potential mechanisms of marked hyperoxaluria not due to primary hyperoxaluria I or II.

Authors
  • 1
Type
Published Article
Journal
Kidney international
Publication Date
Volume
62
Issue
2
Pages
392–400
Identifiers
PMID: 12110000
Source
Medline

Abstract

These patients illustrate a novel form of hyperoxaluria and urolithiasis, without excess Diet(ox), enteric hyper-absorption, or hepatic AGT, GR/HPR deficiency. Alterations in pathways of oxalate synthesis, in liver or kidney, or in renal tubular oxalate handling are possible explanations. The affected sibling pair suggests an inherited basis.

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