Possible involvement of minor lysophospholipids in the increase in plasma lysophosphatidic acid in acute coronary syndrome.

Makoto Kurano; Akiko Suzuki; Asuka Inoue; Yasunori Tokuhara; Kuniyuki Kano; Hirotaka Matsumoto; Koji Igarashi; Ryunosuke Ohkawa; Kazuhiro Nakamura; Tomotaka Dohi; Katsumi Miyauchi; Hiroyuki Daida; Kazuhisa Tsukamoto; Hitoshi Ikeda; Junken Aoki; Yutaka Yatomi

doi:10.1161/ATVBAHA.114.304748

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Possible involvement of minor lysophospholipids in the increase in plasma lysophosphatidic acid in acute coronary syndrome.

Authors

Kurano, Makoto
Suzuki, Akiko
Inoue, Asuka
Tokuhara, Yasunori
Kano, Kuniyuki
Matsumoto, Hirotaka
Igarashi, Koji
Ohkawa, Ryunosuke
Nakamura, Kazuhiro
Dohi, Tomotaka
Miyauchi, Katsumi
Daida, Hiroyuki
Tsukamoto, Kazuhisa
Ikeda, Hitoshi
Aoki, Junken
Yatomi, Yutaka

Type

Published Article

Journal

Arteriosclerosis Thrombosis and Vascular Biology

Publisher

Ovid Technologies Wolters Kluwer -American Heart Association

Publication Date

Feb 01, 2015

Volume

35

Issue

2

Pages

463–470

Identifiers

DOI: 10.1161/ATVBAHA.114.304748

PMID: 25425621

Source

Medline

Keywords

License

Unknown

Abstract

Specific LPA species, especially long-chain unsaturated LPA, were elevated in ACS patients, along with the corresponding minor lysophospholipids. The elevation of these LPA species might be mainly caused by presently unidentified LPA-producing pathway(s). Minor lysophospholipids might be involved in the generation of LPA, especially 22:6 LPA, and in the pathogenesis of ACS.

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine. This record was last updated on 07/05/2016 and may not reflect the most current and accurate biomedical/scientific data available from NLM. The corresponding record at NLM can be accessed at https://www.ncbi.nlm.nih.gov/pubmed/25425621

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