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The phosphoinositide 3 '-kinase p110 delta modulates contractile protein production and IL-6 release in human airway smooth muscle

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Publication Date
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UTS Institutional Repository
Keywords
  • Biochemistry & Molecular Biology
  • Muscle, Smooth
  • Lung
  • Cells, Cultured
  • Humans
  • Asthma
  • Pulmonary Disease, Chronic Obstructive
  • Benzamides
  • Dioxoles
  • Morpholines
  • Chromones
  • Adenine
  • Quinazolines
  • Contractile Proteins
  • Interleukin-6
  • Gene Expression Regulation
  • Adult
  • Aged
  • Aged, 80 And Over
  • Middle Aged
  • Tissue Donors
  • Female
  • Male
  • Transforming Growth Factor Beta1
  • Phosphatidylinositol 3-Kinases
  • Adenine
  • Adult
  • Aged
  • Aged, 80 And Over
  • Asthma
  • Benzamides
  • Cells, Cultured
  • Chromones
  • Class I Phosphatidylinositol 3-Kinases
  • Contractile Proteins
  • Dioxoles
  • Female
  • Gene Expression Regulation
  • Humans
  • Interleukin-6
  • Lung
  • Male
  • Middle Aged
  • Morpholines
  • Muscle, Smooth
  • Phosphatidylinositol 3-Kinases
  • Pulmonary Disease, Chronic Obstructive
  • Quinazolines
  • Tissue Donors
  • Transforming Growth Factor Beta1
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Abstract

Transforming growth factor (TGF) beta 1 increases pro-inflammatory cytokines and contractile protein expression by human airway smooth muscle (ASM) cells, which could augment airway inflammation and hyperresponsiveness. Phosphoinositide 3' kinase (PI3K)

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