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A new metabolic mechanism for absence of pain in patients with silent myocardial ischemia.

Authors
Type
Published Article
Journal
Archives of Medical Research
0188-4409
Publisher
Elsevier
Publication Date
Volume
46
Issue
2
Pages
127–132
Identifiers
DOI: 10.1016/j.arcmed.2015.01.003
PMID: 25616839
Source
Medline
Keywords
  • Chest Pain
  • Lactate
  • Silent Ischemia

Abstract

The novel finding of this study is that the major difference in metabolism during SMI and angina pectoris is in the state of lactate production, which is absent during SMI and present during angina. We assume that lactate is the stimulus of cardiac ischemic pain and when its level increases, it stimulates pain receptors leading to chest pain.

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