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Mechanism of inducible regional dysfunction during dipyridamole stress.

Authors
Type
Published Article
Journal
Circulation
1524-4539
Publisher
Ovid Technologies Wolters Kluwer -American Heart Association
Publication Date
Volume
106
Issue
1
Pages
112–117
Identifiers
PMID: 12093779
Source
Medline
License
Unknown

Abstract

Increased myocardial oxygen demand resulting from hypotension and reflex tachycardia unmasking a reduced endocardial MBF reserve is the primary mechanism of dipyridamole-induced regional dysfunction in chronic coronary artery disease.

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