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Limited proliferation capacity of aortic intima resident macrophages requires monocyte recruitment for atherosclerotic plaque progression

Authors
  • Williams, Jesse W.1, 1, 2
  • Zaitsev, Konstantin3
  • Kim, Ki-Wook2, 4
  • Ivanov, Stoyan2, 5
  • Saunders, Brian T.2
  • Schrank, Patricia R.1, 1
  • Kim, Kyeongdae6
  • Elvington, Andrew2
  • Kim, Seung Hyeon4
  • Tucker, Christopher G.1, 1
  • Wohltmann, Mary2
  • Fife, Brian T.1, 1
  • Epelman, Slava2, 7
  • Artyomov, Maxim N.2
  • Lavine, Kory J.2
  • Zinselmeyer, Bernd H.2
  • Choi, Jae-Hoon6
  • Randolph, Gwendalyn J.2
  • 1 University of Minnesota Medical School, Minneapolis, MN, USA , Minneapolis (United States)
  • 2 Washington University School of Medicine, Saint Louis, MO, USA , Saint Louis (United States)
  • 3 ITMO University, Saint Petersburg, Russia , Saint Petersburg (Russia)
  • 4 University of Illinois College of Medicine, Chicago, IL, USA , Chicago (United States)
  • 5 Centre Méditerranéen de Médecine Moléculaire (C3M), Université Côte d’Azur, Nice, France , Nice (France)
  • 6 Research Institute of Natural Sciences, Hanyang University, Seoul, Republic of Korea , Seoul (South Korea)
  • 7 University of Toronto, Toronto, Ontario, Canada , Toronto (Canada)
Type
Published Article
Journal
Nature Immunology
Publisher
Springer Nature
Publication Date
Sep 07, 2020
Volume
21
Issue
10
Pages
1194–1204
Identifiers
DOI: 10.1038/s41590-020-0768-4
Source
Springer Nature
License
Yellow

Abstract

Williams and colleagues investigate the origin, dynamics and transcriptional profiles of aortic intima macrophages during atherosclerosis disease progression.

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