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Interleukin-1 and tumor necrosis factor-α trigger restriction of hepatitis B virus infection via a cytidine deaminase activation-induced cytidine deaminase (AID).

Authors
Type
Published Article
Journal
Journal of Biological Chemistry
1083-351X
Publisher
American Society for Biochemistry and Molecular Biology
Publication Date
Volume
288
Issue
44
Pages
31715–31727
Identifiers
DOI: 10.1074/jbc.M113.501122
PMID: 24025329
Source
Medline
Keywords
  • Aid
  • Apobec3G
  • Deaminase
  • Hbv
  • Heparg
  • Innate Immunity
  • Interferon
  • Interleukin
  • Tumor Necrosis Factor (Tnf)
  • Virus

Abstract

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