Identification of transport abnormalities in duodenal mucosa and duodenal enterocytes from patients with cystic fibrosis.

V S Pratha; D L Hogan; B A Martensson; J Bernard; R Zhou; J I Isenberg

Identification of transport abnormalities in duodenal mucosa and duodenal enterocytes from patients with cystic fibrosis.

Authors

Pratha, V S
Hogan, D L
Martensson, B A
Bernard, J
Zhou, R
Isenberg, J I

Type

Published Article

Journal

Gastroenterology

Publisher

Elsevier

Publication Date

Jun 01, 2000

Volume

118

Issue

6

Pages

1051–1060

Identifiers

PMID: 10833480

Source

Medline

License

Unknown

Abstract

These findings implicate CFTR in NL duodenal alkaline transport and its absence in CF. Although duodenal bicarbonate secretion is impaired in CF tissues, alternate pathway(s) likely exist that can be activated by carbachol and STa.

From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine. This record was last updated on 07/03/2016 and may not reflect the most current and accurate biomedical/scientific data available from NLM. The corresponding record at NLM can be accessed at https://www.ncbi.nlm.nih.gov/pubmed/10833480

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