hMOF was overexpressed in human NSCLC and was a predictor of poor survival. hMOF-mediated Nrf2 acetylation and nuclear retention are essential for anti-oxidative and anti-drug responses. hMOF may provide a therapeutic target for the treatment of NSCLC.
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The corresponding record at NLM can be accessed at https://www.ncbi.nlm.nih.gov/pubmed/24571482