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Glutaredoxin 2 (Grx2) gene deletion induces early onset of age-dependent cataracts in mice.

Authors
  • 1
  • 2
  • 3
  • 4
  • 5
  • 1 From the School of Veterinary Medicine and Biomedical Sciences, Redox Biology Center, University of Nebraska-Lincoln, Lincoln, Nebraska 68583, the Department of Pharmaceutical Sciences, College of Pharmacy, University of North Texas Health Science Center, Fort Worth, Texas 76107.
  • 2 From the School of Veterinary Medicine and Biomedical Sciences, Redox Biology Center, University of Nebraska-Lincoln, Lincoln, Nebraska 68583, the Department of Ophthalmology, Eye Center of the 2nd Affiliated Hospital, Medical College of Zhejiang University, Hangzhou 310009, China.
  • 3 the Department of Biochemistry and Molecular Biology, Oregon Health and Science University, Portland, Oregon 97239.
  • 4 the Institute of Environment Health Sciences, Wayne State University, Detroit, Michigan 48201, and.
  • 5 From the School of Veterinary Medicine and Biomedical Sciences, Redox Biology Center, University of Nebraska-Lincoln, Lincoln, Nebraska 68583, the Department of Ophthalmology, University of Nebraska Medical Center, Omaha, Nebraska 698583 [email protected]
Type
Published Article
Journal
Journal of Biological Chemistry
1083-351X
Publisher
American Society for Biochemistry and Molecular Biology
Publication Date
Volume
289
Issue
52
Pages
36125–36139
Identifiers
DOI: 10.1074/jbc.M114.620047
PMID: 25362663
Source
Medline
Keywords
  • Age-Related Cataract
  • Electron Transport System
  • Glutaredoxin 2
  • Oxidative Stress
  • α-Crystallin Protein

Abstract

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