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The enhanced pressor response in type 2 diabetes is not based upon a generalized increase in vascular responsiveness.

Authors
Type
Published Article
Journal
Cardiovascular research
Publication Date
Volume
38
Issue
1
Pages
206–214
Identifiers
PMID: 9683923
Source
Medline
License
Unknown

Abstract

Non-obese normotensive type 2 patients have an increased pressor response to NA, which is not based upon a defect in skeletal muscle resistance arterioles, peripheral veins, or a defect in the baroreceptor system. Therefore, in type 2 diabetes the noradrenergic responsiveness of other vascular beds, such as the splanchnic or renal, must be enhanced.

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