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Enhanced gene expression of chemokines and their corresponding receptors in mononuclear blood cells in chronic heart failure--modulatory effect of intravenous immunoglobulin.

Authors
  • Damås, J K
  • Gullestad, L
  • Aass, H
  • Simonsen, S
  • Fjeld, J G
  • Wikeby, L
  • Ueland, T
  • Eiken, H G
  • Frøland, S S
  • Aukrust, P
Type
Published Article
Journal
Journal of the American College of Cardiology
Publisher
Elsevier
Publication Date
Jul 01, 2001
Volume
38
Issue
1
Pages
187–193
Identifiers
PMID: 11451272
Source
Medline
License
Unknown

Abstract

Our results further support a pathogenic role for chemokines in CHF and suggest that IVIg may represent a novel therapeutic approach, with the potential to improve LVEF in patients with CHF, possibly by modulatory effects on the chemokine network.

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