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Endothelin B receptor-mediated encephalopathic events in mouse sepsis model.

Authors
  • Naito, Yusuke1
  • Yoshioka, Kento2
  • Tanaka, Kensuke1
  • Tatsumi, Koichiro3
  • Kimura, Sadao2
  • Kasuya, Yoshitoshi4
  • 1 Department of Biochemistry and Molecular Pharmacology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan; Department of Respirology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan. , (Japan)
  • 2 Department of Biochemistry and Molecular Pharmacology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan. , (Japan)
  • 3 Department of Respirology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan. , (Japan)
  • 4 Department of Biochemistry and Molecular Pharmacology, Graduate School of Medicine, Chiba University, Chiba 260-8670, Japan. Electronic address: [email protected] , (Japan)
Type
Published Article
Journal
Life sciences
Publication Date
Nov 24, 2014
Volume
118
Issue
2
Pages
340–346
Identifiers
DOI: 10.1016/j.lfs.2014.03.012
PMID: 24657829
Source
Medline
Keywords
License
Unknown

Abstract

We demonstrated that BQ788 could protect the brain from the following sepsis-associated pathophysiological output: neural cell death, inflammatory response and the Hans Selye's environmental stress reaction.

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