C pneumoniae impairs arterial endothelial function, and the NO pathway is principally involved. Cyclooxygenase-dependent vasoconstricting products may also account for the infection-induced impaired relaxation. These findings further support the role of C pneumoniae infection in atherosclerosis development.
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The corresponding record at NLM can be accessed at https://www.ncbi.nlm.nih.gov/pubmed/10961970