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The effect of applied chest compression force on systemic arterial pressure and end-tidal carbon dioxide concentration during CPR in human beings.

Authors
  • Ornato, J P
  • Levine, R L
  • Young, D S
  • Racht, E M
  • Garnett, A R
  • Gonzalez, E R
Type
Published Article
Journal
Annals of Emergency Medicine
Publisher
Elsevier
Publication Date
Jul 01, 1989
Volume
18
Issue
7
Pages
732–737
Identifiers
PMID: 2500044
Source
Medline
License
Unknown

Abstract

Twelve adult (nine men and three women) cardiac arrest patients were studied as they received CPR by a computerized Thumper to determine the influence of the applied chest compression force on blood flow (as assessed by the end-tidal carbon dioxide concentration) and arterial pressure. At the end of a resuscitation when the decision was made by the senior physician to cease resuscitative efforts, the applied force on the CPR Thumper was decreased from 140 to 0 pound-force (lbf) in 20-lbf increments at 30-second intervals. Radial artery cutdown blood pressure and end-tidal carbon dioxide (ETCO2) were recorded continuously. Arterial systolic blood pressure was linearly related (r = .59, P less than .0001) to applied force (systolic blood pressure, 31 +/- 6 mm Hg at 20 lbf to 60 +/- 7 mm Hg at 140 lbf). ETCO2 (r = .42, P less than .0001) was also linearly related to applied force (ETCO2, 0.7 +/- 0.1% at 20 lbf to 1.5 +/- 0.2% at 140 lbf). Diastolic pressure did not change significantly with change in applied force (17 +/- 2 mm Hg from 20 to 140 lbf). Our findings indicate that higher compression force than that currently recommended may improve arterial systolic pressure and flow in human beings receiving closed-chest compression during CPR.

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