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Defective autophagosome trafficking contributes to impaired autophagic flux in coronary arterial myocytes lacking CD38 gene.

Authors
  • Zhang, Yang
  • Xu, Ming
  • Xia, Min
  • Li, Xiang
  • Boini, Krishna M
  • Wang, Mi
  • Gulbins, Erich
  • Ratz, Paul H
  • Li, Pin-Lan
Type
Published Article
Journal
Cardiovascular Research
Publisher
Oxford University Press
Publication Date
Apr 01, 2014
Volume
102
Issue
1
Pages
68–78
Identifiers
DOI: 10.1093/cvr/cvu011
PMID: 24445604
Source
Medline
Keywords
License
Unknown

Abstract

Taken together, these results suggest that CD38 plays a critical role in autophagosome trafficking and fusion with lysosomes, thus controlling autophagic flux in CAMs under atherogenic stimulation.

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