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Defective autophagosome trafficking contributes to impaired autophagic flux in coronary arterial myocytes lacking CD38 gene.

Authors
Type
Published Article
Journal
Cardiovascular Research
1755-3245
Publisher
Oxford University Press
Publication Date
Volume
102
Issue
1
Pages
68–78
Identifiers
DOI: 10.1093/cvr/cvu011
PMID: 24445604
Source
Medline
Keywords
License
Unknown

Abstract

Taken together, these results suggest that CD38 plays a critical role in autophagosome trafficking and fusion with lysosomes, thus controlling autophagic flux in CAMs under atherogenic stimulation.

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