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C5a receptor enables participation of mast cells in immune complex arthritis independently of Fcγ receptor modulation.

Authors
  • Nigrovic, Peter A
  • Malbec, Odile
  • Lu, Bao
  • Markiewski, Maciej M
  • Kepley, Chris
  • Gerard, Norma
  • Gerard, Craig
  • Daëron, Marc
  • Lee, David M
Type
Published Article
Journal
Arthritis & Rheumatism
Publisher
Wiley (John Wiley & Sons)
Publication Date
Nov 01, 2010
Volume
62
Issue
11
Pages
3322–3333
Identifiers
DOI: 10.1002/art.27659
PMID: 20662064
Source
Medline
License
Unknown

Abstract

Stimulation via C5aR is required to unleash the proinflammatory activity of synovial mast cells in immune complex arthritis, albeit via a mechanism that is distinct from C5a-modulated expression of FcγR.

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