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Auditory brainstem function in microcephaly related to Zika virus infection.

Authors
  • Marques Abramov, Dimitri1
  • Saad, Tania1
  • Gomes-Junior, Saint-Clair1
  • de Souza E Silva, Daniel1
  • Araújo, Izabel1
  • Lopes Moreira, Maria Elizabeth1
  • Lazarev, Vladimir V2
  • 1 From the Laboratory of Neurobiology and Clinical Neurophysiology (D.M.A., T.S., D.d.S.e.S., I.A., V.V.L.) and Unit of Clinical Research (S.-C.G.-J., M.E.L.M.), National Institute of Women, Children and Adolescents, Health Fernandes Figueira, Oswaldo Cruz Foundation (FIOCRUZ), Ministry of Health, Rio de Janeiro, Brazil. , (Brazil)
  • 2 From the Laboratory of Neurobiology and Clinical Neurophysiology (D.M.A., T.S., D.d.S.e.S., I.A., V.V.L.) and Unit of Clinical Research (S.-C.G.-J., M.E.L.M.), National Institute of Women, Children and Adolescents, Health Fernandes Figueira, Oswaldo Cruz Foundation (FIOCRUZ), Ministry of Health, Rio de Janeiro, Brazil. [email protected] , (Brazil)
Type
Published Article
Journal
Neurology
Publisher
Ovid Technologies (Wolters Kluwer) - American Academy of Neurology
Publication Date
Feb 13, 2018
Volume
90
Issue
7
Identifiers
DOI: 10.1212/WNL.0000000000004974
PMID: 29352094
Source
Medline
License
Unknown

Abstract

These results are consistent with the functional normality of the brainstem structure and its lack of correlation with microcephaly, suggesting that the disruption produced by the ZV infection does not act in the cell proliferation phase, but mostly in the processes of neuronal migration and differentiation in the telencephalon.

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