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Atrial Substrate and Triggers of Paroxysmal Atrial Fibrillation in Patients With Obstructive Sleep Apnea.

Authors
  • Anter, Elad1
  • Di Biase, Luigi2
  • Contreras-Valdes, Fernando M2
  • Gianni, Carola2
  • Mohanty, Sanghamitra2
  • Tschabrunn, Cory M2
  • Viles-Gonzalez, Juan F2
  • Leshem, Eran2
  • Buxton, Alfred E2
  • Kulbak, Guy2
  • Halaby, Rim N2
  • Zimetbaum, Peter J2
  • Waks, Jonathan W2
  • Thomas, Robert J2
  • Natale, Andrea2
  • Josephson, Mark E2
  • 1 From the Cardiovascular Division, Department of Medicine, Harvard-Thorndike Electrophysiology Institute (E.A., F.M.C.-V., C.M.T., E.L., A.E.B., G.K., R.N.H., P.J.Z., J.W.W., M.E.J.) and Sleep Disorders Clinic, Departments of Medicine and Neurology (R.J.T.), Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA; Texas Cardiac Arrhythmia Institute, St. David's Medical Center, Austin (L.D.B., C.G., S.M., A.N.); and Electrophysiology Section, Cardiovascular Division, University of Miami Miller School of Medicine, FL (J.F.V.-G.). [email protected] , (Israel)
  • 2 From the Cardiovascular Division, Department of Medicine, Harvard-Thorndike Electrophysiology Institute (E.A., F.M.C.-V., C.M.T., E.L., A.E.B., G.K., R.N.H., P.J.Z., J.W.W., M.E.J.) and Sleep Disorders Clinic, Departments of Medicine and Neurology (R.J.T.), Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA; Texas Cardiac Arrhythmia Institute, St. David's Medical Center, Austin (L.D.B., C.G., S.M., A.N.); and Electrophysiology Section, Cardiovascular Division, University of Miami Miller School of Medicine, FL (J.F.V.-G.). , (Israel)
Type
Published Article
Journal
Circulation Arrhythmia and Electrophysiology
Publisher
Ovid Technologies Wolters Kluwer -American Heart Association
Publication Date
Nov 01, 2017
Volume
10
Issue
11
Identifiers
DOI: 10.1161/CIRCEP.117.005407
PMID: 29133380
Source
Medline
Keywords
License
Unknown

Abstract

OSA is associated with structural and functional atrial remodeling and increased incidence of extra-PV triggers. Elimination of these triggers resulted in improved arrhythmia-free survival.

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