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Association of tumor necrosis factor receptor type II polymorphism 196R with Systemic lupus erythematosus in the Japanese: molecular and functional analysis.

Authors
Type
Published Article
Journal
Arthritis and rheumatism
Publication Date
Volume
44
Issue
12
Pages
2819–2827
Identifiers
PMID: 11762942
Source
Medline
License
Unknown

Abstract

These results suggest that 196R TNFRII, which transduces the signals of TNFalpha more effectively than does 196M TNFRII, is involved in the pathogenesis of SLE.

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