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Advanced glycation end products depress function of endothelial progenitor cells via p38 and ERK 1/2 mitogen-activated protein kinase pathways.

Authors
  • Sun, Chengbo
  • Liang, Chun
  • Ren, Yusheng
  • Zhen, Yi
  • He, Zhiqing
  • Wang, Hua
  • Tan, Hongbing
  • Pan, Xiaoming
  • Wu, Zonggui
Type
Published Article
Journal
Basic research in cardiology
Publication Date
Jan 01, 2009
Volume
104
Issue
1
Pages
42–49
Identifiers
DOI: 10.1007/s00395-008-0738-8
PMID: 18622638
Source
Medline
License
Unknown

Abstract

AGE-HSA could promote atherosclerosis by upregulating EPCs RAGE expressions and promoting EPCs apoptosis via p38, ERK MAPK pathways, activation of NF-kappaB might also play a role in this process.

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