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UV-induced reversion of a temperature-sensitive late mutant of simian virus 40 to a wild-type phenotype.

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PMC
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  • Research Article

Abstract

Irradiation of a simian virus 40 temperature-sensitive late mutant before infection of CV1 cells produces viruses that are phenotypically wild-type revertants. This probably occurs through suppression rather than true reversion because the revertants are produced at relatively high frequency and still have some properties of the original temperature-sensitive virus.

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