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Virulence mechanisms and host specificity of gall-formingPantoea agglomerans

Authors
Journal
Trends in Microbiology
0966-842X
Publisher
Elsevier
Publication Date
Volume
15
Issue
12
Identifiers
DOI: 10.1016/j.tim.2007.10.009
Disciplines
  • Biology

Abstract

Pantoea agglomerans has been transformed from a commensal bacterium associated with many plants into a host-specific gall-forming pathogen by acquiring a plasmid-borne pathogenicity island. This pathogenicity island harbors the hrp/ hrc gene cluster, in addition to genes encoding type III effector proteins, biosynthesis of the phytohormones indole-3-acetic acid and cytokinin, multiple diverse insertion sequences and pseudogenes. This review describes a unique model for understanding the emergence of new pathogens or new pathogenic variants, offering an insight into the function of type III effectors in host specificity and the evolution of a pathogen into pathovars. It also addresses the primary role of type III effectors in gall initiation as compared with a secondary role of phytohormones secreted by the pathogen.

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