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Amyotrophic lateral sclerosis: new genes, new models, and new mechanisms

Authors
Journal
F1000 Biology Reports
1757-594X
Publisher
"Faculty of 1000, Ltd."
Publication Date
Volume
3
Identifiers
DOI: 10.3410/b3-18
Keywords
  • Review Article

Abstract

Research aimed at understanding amyotrophic lateral sclerosis (ALS) has seen exceptional growth in the past few years. New genes, new models, and new mechanisms have not only improved our understanding, but also contributed to the increasing complexity of ALS pathogenesis. The focus of this piece is to highlight some of the more notable developments in the field and to encourage a re-appreciation for the superoxide dismutase 1 (SOD1) mouse models.

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